Alaskan Malamute Health
Every malamute owner and prospective buyer should be aware of the health concerns specific to Alaskan Malamutes. The potential medical conditions listed in this overview can vary greatly in severity or not affect your dog at all. This is not a complete listing and does not include common health issues such as canine diseases.
A more detailed explanation and updated health information is available through good medical reference guides or from your veterinarian. Additional Malamute information may be found on the Alaskan Malamute Health website. If you suspect that your dog is effected by any of these conditions, or any health ailment, please consult your veterinarian.
Medical conditions and disorders found in this overview...
Bloat (Gastric Dilation and Gastric Torsion):
Bloat is a general term refering to gastric dilation (stomach expands) &/or gastric torsion (stomach twists).
Gastric Dilation is a build up of air or stomach contents that cannot be passed through the intestines, or expelled by "burping"/ vomiting. The internal blockage may last hours or even days. Severe dilation is LIFE THREATENING requiring immediate medical attention and may lead to torsion (twisting) of the stomach.
Gastric Torsion is the full or partial twisting of the stomach, cutting off blood supplies to other organs. Complete torsion may cause a painful death in a matter of minutes! Rapid diagnosis and treatment by a veterinarian is essential to save the life of your dog. Dogs that have "bloated" and survived are at high risk for a recurrence of this condition.
The exact cause of bloat is not actually known. It does seem to occur mostly within a few hours of eating or drinking, especially if the dog has been exercised shortly after the meal or is a rapid "gulping" eater. Other causes of bloat are (but not limited to) overeating, intestinal blockage by foreign materials, traumatic injury, or physical stress (whelping, vomiting, etc).
Bloat is not known to be genetic. However, a tendency toward the condition may be inheritable, as well as specific intestinal defects. Elevating the food bowl may worsen bloat occurance by allowing the dog to gulp more air as it eats.
Indications of bloat include abdominal distention (tight/"bloated" stomach), restlessness, lethargy, excessive salivation (drooling) or panting, retching without actually vomiting, and/or "watery" diarrhea (liquids pass, but solids do not).
Condition where the lens of the eye becomes clouded or opaque, impairing the vision. The degree of vision loss depends upon the size and location of the cataract within the eye. Cataracts may also cause a lens protein to leak into the eye, resulting in an immune reaction and inflammation of the eye. Surgical replacement of the affected lens is the only method to restore vision.
Cataracts can be a result of old age, disease (such as diabetis), trauma to the eye, or be congenital (before birth). Congenital cataracts may be inherited and usually do not lead to total blindness as they do not change in size. They do, however, present a handicap to a growing pup depending on severity.
Coat disorder characterized by the breaking and eventual loss of the guard coat. The hair does not grow back and will eventually give the affected dogs body a "woolly lamb" appearance. Males are usually affected, but cases of affected females or only a loss of undercoat have been reported. It is suspected (but not yet proven) to be found within family lines and sex related, or caused by mineral or thyroid hormone imbalance, or some combination.
Coat funk appears to be a problem with the hair follicle cycle where the normal cycle of shed & regrowth halts. The hair becomes brittle with age, coat breaks off and lost hair is not replaced. Lab tests such as thyroid level and skin scrapings will appear normal.
Symptoms of coat funk first appear around 2-3 years of age, but may not attract much concern by owners until the severity increases. Initial signs of the disorder are coat "wear" or breakage around the collar, tail, and hair stress points such as the haunches and buttocks. Eventually this pattern of broken coat will spread to the rest of the body.
Neutering/spaying the affected dog may cause the hair follicles to act normally for 1-2 cycles before halting again. In very mild cases, it is possible that the affected dog may not show renewed signs of coat funk until middle or late age. Hormone therapy or dietary change may help control the symptoms, but is not usually considered curative treatment.
Affected dogs appear normal in all other respects, and can lead full and happy lives. However, care must be given to protect them from the elements... cold or wet weather, excessive exposure to sun or wind, etc.
Coat Funk (Coat Odor)
Common or slang name for a coat that has a strong chronic odor. Characterized by a "sour" smell and returns within a few days of bathing. Little is known about the cause of this condition, but may be linked in some instances to a border line thyroid problem. Other suspected causes are vitamin or mineral deficiencies and allergies. Dietary change or vitamin/mineral therapy may help.
Occasional (non chronic) strong coat odor or "funk" can be caused by a fungal or bacterial growth in the undercoat. In these instances, the undercoat has become wet from bathing, swimming, etc and was either improperly or incompletely dried.
Day Blindness (Hemeralopia)
A retinal disorder causing an inability to see objects and determine distances during exposure to daylight. An affected dog may have partial or normal vision under low light conditions such as night/evening, dusk/dawn, when indoors, or during overcast days. The severity of the disorder varies in each dog affected.
Day blindness is genetically inherited through a recessive trait. It does not worsen over time and may be detected in puppies less than two months old. There is no known effective treatment at this time. Affected dogs should be monitored and their activities restricted during daylight hours.
A recessive genetic condition involving the development of the growth plates in the legs, resulting in stunted or deformed growth. It is most noticeable in the forelegs which can become short, squat, "knobby", and bow inward under the body. An effected dwarf may be barely able to walk or seem almost normal, depending on the severity of the condition.
The Alaskan Malamute Club of America undertook a study and a test-breeding program to eliminate dwarfism. Percentile ratings were given to dogs on the basis of their genetic background. Ratings lower than 6.25% were considered safe to breed, while a higher percentage rate required the dog be test-bred to be proven clear of the condition. Percentile ratings are no longer given and a dog is either considered "clear" or not clear of the condition.
The study was revived in the late 1990s and research begun to isolate a DNA marker to determine the dwarfism potential without performing a test breeding. A blood test to help determind dwarfism is now available.
Elbow Dysplasia (Anconeal Dysplasia)
This condition involves the improper development of the small bones in the elbow, which do not grow together as they should. This results in lameness, poor extension of the elbow, pain and swelling.
The cause of this abnormality is not known. Surgery may alleviate the condition as long as arthritis has not developed in the elbow.
OFA (Orthopedic Foundation for Animals) certifies dogs on a clear or not clear basis using x-rays, but few dogs are tested for this condition prior to symptoms.
A disorder of the brain caused by abnormal electrical bursts and characterized by seizures. Other symptoms may include rigidity, anxiety or hysteria, disorientation, unconsciousness, vocal outbursts, salivation/drooling, and loss of bladder or bowel control.
The cause may be hereditary, trauma to the head or nervous system, chemical imbalance, or environmental exposure to chemicals. Seizures may occur only once or twice, or there may be several attacks at varying intervals anywhere from several minutes to many months. Recurrent attacks may be of decreased intervals and increased duration.
A hereditary condition resulting in the improper development of the hip joint. The socket of the joint will be deformed or too shallow, allowing the rounded end of the thigh bone to separate from the socket. In most cases the rounded "ball" end of the thigh bone will be abnormally flattened and the "neck" of the bone may show signs of thickening. This condition can also be caused from hip ligaments &/or muscles not having enough control over the hip during movement (i.e., trauma to the hip area during development).
Most breeds are at risk and especially the larger dogs. This is probably due to the greater weight of the body and the associated greater stress to the joints. Dogs with hip dysplasia are born with normal hips, but the condition will usually manifest itself within the first two years. The more severe the hip joint abnormality... generally the sooner it will become apparent.
Hip dysplasia may vary from mildly abnormal development to complete hip dislocation. The severity of the condition may also be influenced by too rapid growth, overfeeding (over nutrition), or excessive exercise. It is usually painful and interferes with proper movement and activity levels, depending upon the severity.
Diagnosis is made by X-raying the hip joint. Dogs at least 2 years of age can obtain a hip certification through the OFA (Orthopedic Foundation for Animals), which rates hip joints from "severely dysplastic" to "excellent". It is highly recommended NOT to purchase dogs who are not certified clear of hip dysplasia, puppies or dogs whose parents are not certified clear, or from breeders who do not furnish a copy of the certification upon request.
Treatment depends upon the severity of hip deformity. Mild cases may require the dog to be on a life-long prescription of pain medication. Surgery to reconstruct or replace the hip joint may be required in more severe cases or as the hip joint wears with age. In severely dysplastic cases the dog may require euthanasia (death).
Disorder caused by the deficiency of a thyroid hormone and is marked by a low metabolic rate. Usually caused by the destruction of the thyroid gland from an immune process, atrophy or cancer. Although not known to be inherited, the general genetic makeup of the dog/breed may be partially responsible for the development of inflammation of the thyroid gland, an immune mediated condition. Hypothyroidism is the most common of hormone disorders in dogs, and Malamutes are one of the breeds that appear to be at increased risk.
Some signs of hypothyroidism include mental dullness, avoidance/intolerance to exercise, general lethargy, weight gain without increased food intake, slow or poor coordination, seizures, poor coat (dry, dull, loss, slow regrowth) and skin condition (dry/scaley), as well as resproductive problems.
Symptoms may be gradual and subtle, and usually appear between two and six years of age. Treatment consists of hormone replacement therapy, which must continue throughout the dog's life, and recovery to a normal lifestyle is excellent.
(Note 1. Hypothyroidism has been linked to some cases of polyneuropathic conditions. Hypothyroidism associated with polyneuropathies can be difficult to detect due to the lack of normal outward symptoms. Thyroid levels in a simple T4 blood screen may appear relatively normal and diagnosis is usually made by testing T4 levels after use of a thyroid stimulant.)
(Note 2: Hypothyroidism , whether definite or borderline, has been linked to certain types of skin and coat problems such as "coat funk".
(Note 3. Severe hypothyroidism in very young dogs can result in "cretinism". Cretinism is characterized by dull-wittedness, lethargy, short thick bodies with large heads, enlarged thyroid gland, and slow physical development.)
Inflammation of the sensory and motor nerve fibers resulting in nerve degeneration (damage) and progressive muscle weakness. Characterized by gradual onset and slow progression of symptoms. The earliest indications may be a change in voice, difficulty in swallowing, or regurgitation of food. Further signs are uncoordinated movement, palsy (trembling muscles), loss of balance and eventual paralysis of the legs.
It is suspected, but not known, to be hereditary in Malamutes. In these cases only males are affected and their grand sire (the dam's father) may also have been affected. Hypothyroidism, another suspected hereditary condition in Malamutes, may be an underlying culprit in some cases (HT, note 1).
Other known or suspected causes include physical trauma, dysfunctional immune system, drug or chemical toxicity (organophosphates, trichlorethylene, etc), heavy metal toxicity (lead, copper, zinc, etc), metabolic diseases (hypothyroidism, diabetes, etc), and cancer.
Treatment is dependent upon the underlying cause of the condition. Recovery will depend upon the degree of nerve damage involved. The specific cause in many dogs may not be identifiable and no effective therapy available. In cases suspected to be hereditary, most dogs will eventually recover on their own. However, these dogs will not fully recover to their pre-polyneuropathic condition and will require some form of invalid care in the meantime.
Progressive Retinal Atrophy: PRA & CPRA
A hereditary and degenerative condition of the retina which causes impaired vision and slow or incomplete light reaction by the pupil. (Note: The retina is the deepest of three main tissue layers which make up the inner wall at the back of the eye.) Normally the condition appears between the ages of four and eight years old. There are two types of progressive retinal atrophy, general (PRA) and central (CPRA).
PRA is the more common type of retinal atrophy and affects the photoreceptor area of the retina. CPRA is similar to PRA, but affects the retinal layer beneath the photoreceptive area. Symptoms may be subtle at first, including a reluctance to go outside at night, staying near lighted areas or their owner, difficulty in tracking a moving object, reluctance to climb stairs, or misjudging indoor jumps.
Initial onset is characterized by night blindness (poor vision in dim lighting) and normal vision in the daylight. Progression of the disorder eventually leads to loss of day vision and later total blindness. In the final stages the pupil does not react to strong light and is widely dilated. Cataracts are not uncommon. There is no known effective treatment.
Wobbler's Syndrome (Cervical Spondylopathy)
A hereditary condition which is a failure of proper support to the vertebrae area and affects the spinal cord in the hip area. Found mostly in large breeds or sometimes in long backed breeds. This is a fatal condition which usually progresses slowly, but in some cases can cripple a dog in less than a day. Onset is normally between three and twelve months of age. The exact cause is not known, however displacement of vertebrae due to a long neck, overfeeding (over nutrition), and too rapid growth is suspect in influencing the condition.
Characterized by a progressive lack of coordination in the hindquarters due to very weak & unsupportive leg muscles and a palsy-like shaking of the head. As the condition continues, the front quarters become affected and the rear will eventually become completely unsupportive (quadriplegic). The condition is frequently extremely painful.
Diagnosis is by X-ray. Treatment consists of surgery to alleviate displacement/deformity of the vertebrae. Acute cases respond best to surgery, slowly progressive cases the least.
Zinc Responsive Dermatitis:
A scaling skin disease caused by the inability to absorb sufficient zinc amounts from the intestine. This is due either to a genetic defect (sled dog breeds in particular) or to a nutritional imbalance. Some dog food ingredients and food supplements are known to decrease zinc absorption. Among these are calcium, iron, tin, copper and phytates (plant sugar). Calcium is the most commonly used supplement and should not be given to growing puppies for this reason.
Indications of this condition include scaley/crusty skin, itchiness, dull or brittle coat, or hair loss. This may be most noticeable on the face, hocks and elbows. Zinc absorption problems may also play a part in pigment loss. Over supplementing growing puppies or young dogs (especially with calcium) may cause poor appetite, stunted growth, or deformed bones.
Diagnosis is by skin biopsy of the affected area. Treatment is by the use of zinc supplements, either until the condition is alleviated (nutritionally caused) or for the life of the dog (genetic).